Initial optimization and series evolution of diaminopyrimidine inhibitors of interleukin-1 receptor associated kinase 4.

نویسندگان

  • W Michael Seganish
  • William T McElroy
  • R Jason Herr
  • Stephanie Brumfield
  • William J Greenlee
  • James Harding
  • Venukrishnan Komanduri
  • Julius Matasi
  • Koraboina Chandra Prakash
  • Deen Tulshian
  • Jinhai Yang
  • Larry Yet
  • Kristine Devito
  • James Fossetta
  • Charles G Garlisi
  • Daniel Lundell
  • Xiaoda Niu
  • Christopher Sondey
چکیده

IRAK4 plays a key role in TLR/IL-1 signaling. Previous efforts identified a series of aminopyrimidine IRAK4 inhibitors that possess good potency, but modest kinase selectivity. Exploration of substituents at the C-2 and C-5 positions generated compounds that maintained IRAK4 potency and improved kinase selectivity. Additionally, it was found that the pyrimidine core could be replaced with a pyridine and still retain potency and kinase selectivity. The optimization efforts led to compound 26 which had an IRAK4 IC50 of 0.7 nM, an IC50 of 55 nM on THP-1 cells stimulated with LPS, a TLR4 agonist, and greater than 100-fold selectivity versus 96% of a panel of 306 kinases.

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عنوان ژورنال:
  • Bioorganic & medicinal chemistry letters

دوره 25 16  شماره 

صفحات  -

تاریخ انتشار 2015